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Hepatit A Enfeksiyonu Sonrası Gelişen Bir Guillian-Barre Sendromu Olgusu+

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dc.contributor.author Özışık, Handan Işın
dc.contributor.author Kızkın, Sibel
dc.contributor.author Çalışkan, Özden
dc.contributor.author Özcan, Cemal
dc.date.accessioned 2015-04-16T13:09:21Z
dc.date.available 2015-04-16T13:09:21Z
dc.date.issued 2002
dc.identifier.citation Özışık, Handan Işın ;Kızkın, Sibel ; Çalışkan, Özden ; Özcan, Cemal ;İnönü Üniversitesi Tıp Fakültesi Dergisi 9(2) 131-133 (2002) tr_TR
dc.identifier.uri http://www.totmdergisi.org/articles/2002/volume9/issue2/2002_9_2_12.pdf
dc.identifier.uri http://hdl.handle.net/11616/1875
dc.description İnönü Üniversitesi Tıp Fakültesi Dergisi 9(2) 131-133 (2002) tr_TR
dc.description.abstract Guillian-Barre Syndrome (GBS) is often preceded by an infectious disease. A case of GBS after hepatitis A (HA) is extremely rare, even though hepatitis A is common. In a review of case reports the clinical features of GBS following HA could be sumarized as follows: 1-Most of the patients are men 2- GBS develops within 14 days after onset of HA 3- Facial nerve and bulbus are effected frequently 4- Proprioception is likely impaired in addition to superficial sensation 5- Mechanic ventilation is often requirement 6- The protein content of the cerebrospinal fluid is usually high 7- The outcome of neuropathic symptoms is uniformly good, regardless of the degree of liver dysfunction as evaluated on the basis of alanine aminotransferase levels 8- Patient is frequently recover without sequelaw 9- ENMG demonstrate demyelinations. These findings indicate that GBS following HA essentially does not differ from typical GBS. tr_TR
dc.description.abstract Guillian-Barre Syndrome (GBS) is often preceded by an infectious disease. A case of GBS after hepatitis A (HA) is extremely rare, even though hepatitis A is common. In a review of case reports the clinical features of GBS following HA could be sumarized as follows: 1-Most of the patients are men 2- GBS develops within 14 days after onset of HA 3- Facial nerve and bulbus are effected frequently 4- Proprioception is likely impaired in addition to superficial sensation 5- Mechanic ventilation is often requirement 6- The protein content of the cerebrospinal fluid is usually high 7- The outcome of neuropathic symptoms is uniformly good, regardless of the degree of liver dysfunction as evaluated on the basis of alanine aminotransferase levels 8- Patient is frequently recover without sequelaw 9- ENMG demonstrate demyelinations. These findings indicate that GBS following HA essentially does not differ from typical GBS. tr_TR
dc.language.iso tur tr_TR
dc.publisher İnönü Üniversitesi Tıp Fakültesi Dergisi tr_TR
dc.rights Attribution 3.0 United States *
dc.rights.uri http://creativecommons.org/licenses/by/3.0/us/ *
dc.subject Hepatit A tr_TR
dc.subject Guillian-Barre Sendromu tr_TR
dc.subject GBS Klinik Bulguları tr_TR
dc.title Hepatit A Enfeksiyonu Sonrası Gelişen Bir Guillian-Barre Sendromu Olgusu+ tr_TR
dc.title.alternative A Case Of Guillian-Barre Syndrome After Acute Viral Hepatitis A tr_TR
dc.type Article tr_TR


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