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    Peripheral facial paralysis during the COVID-19 pandemic
    (2022) Aydın, Şükrü; Fırat Koca, Çiğdem; Çelik, Turgut; Kelleş, Mehmet; Yaşar, Şeyma
    The mechanism surrounding idiopathic peripheral facial nerve paralysis remains unclear, though viral infections and even immunizations have been suspected of its origin. Thus, the relationship between COVID-19 and facial paralysis should be studied. With this research, we aimed to investigate the characteristics of facial paralysis during the COVID-19 illness as well as the relationship between facial paralysis and COVID-19, the length of time needed for recovery, concurrence with COVID-19 infection, and whether facial paralysis is a late complication or initial symptom of the disease. Forty-five patients thought to have had idiopathic peripheral facial paralysis were included in the study. Pure tone audiometry, COVID-19 PCR tests, and contrast-enhanced ear MRIs were performed on all participants. A standard prednisolone treatment protocol was followed. Participants were monitored for one month; we recorded whether they had COVID-19 previously, initially, or contracted it within the one-month testing period. At the same time, facial paralysis recovery rates were recorded and used in statistical analyses. PCR test at initial admission was reported as positive for COVID-19 in only one participant (2.2%). We discovered an improvement delay regarding facial paralysis in participants who had had COVID-19 previously (p<0.001). Prednisolone therapy used for peripheral facial paralysis did not pose an additional risk for COVID-19. Having had COVID-19 previously may cause delayed recovery of peripheral facial paralysis. Peripheral facial paralysis may be both a late manifestation as well as an early symptom of COVID-19.
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    Öğe
    The investigation of vestibulary system in patients with thyroid dysfunctions
    (2024) Yaşar, Şeyma; Aydin, Sukru; Koca, Çiğdem Fırat; Çelik, Turgut; Kelleş, Mehmet; Keskin, Lezan
    A relationship has been detected among thyroid dysfunctions and vestibular pathologies. Our aim was to evaluate the vestibular system in cases with thyroid dysfunctions. Totally 71 patients between the ages of 20-50 with thyroid dysfunctions (35 hypothyroidic and 36 hyperthyroidic) and 36 participants with normal thyroid functions were included. Patients were also allocated as two groups based on antibody positivity. Pure tone audiometry and Vestibular Evoked Myogenic Potential tests were performed for all participants. In the left ear there was a numerically significance in the hypothyroids at 4000 and 8000 Hz (p=0.020 and p=0.014). In the right ear p13 and n23 latencies differed among the groups, (p=0.004 and p=0.001). The p13-n23 and n10-p15 amplitudes were decreased in both patient groups (p<0.001) (p=0.015). For the left ear, the p13 an n23 latencies was prolonged in both patient groups (p=0.044) (p=0.002). The p13-n23 amplitude value was distincted numerically significant (p<0.001). The n10-p15 amplitude was longer in the patients (p=0.004). We can not observe a numerically significance among groups according to antibody positivity (p>0.005). Thyroid dysfunctions may lead to subclinical changes on the vestibular system and the saccular and utriculer impairment may be an early indicator of endolymphatic hydrops.