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    Kaposi's varicelliform eruption complicating irritant contact dermatitis
    (Professional Medical Publications, 2012) Sener, Serpil; Bayram, Hatice Gamze; Karincaoglu, Yelda; Senol, Mustafa
    Kaposi's varicelliform eruption (KVE) or eczema herpeticum is a disseminated viral infection superimposed on pre-existing dermatosis. It is frequently caused by Herpes simplex and certain other viruses. It may result on life-threatening viraemia with multiorgan involvement and secondary infections. We report a 34-year old female patient with KVE which is caused by herpes virus developed on irritant contact dermatitis because of its rare occurrence.
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    Prophylactic Antiviral Treatment in Recurrent Herpes Zoster: A Case Report
    (Galenos Publ House, 2011) Bayram, Hatice Gamze; Ozcan, Hamdi; Bayindir, Yasar
    Herpes zoster (HZ) occurs in older ages with activation of varicella-zoster virus (VZV) which persists in a dormant phase within the dorsal root ganglia. The incidence of HZ in immunosuppressed patients is 20-100 times higher and the clinical progress is more severe than in immunocompetent individuals. A 48-year-old man who had been diagnosed with acute myelocytic leukemia type M3 and had been treated with immunosuppressive agents was admitted to our clinic. The patient was clinically diagnosed as having HZ. He was treated with acyclovir 800 mg five times daily for 7 days. In the consecutive three months, he attended our clinic again with similar complaints. The left cervical (C)5, C6 dermatomes were involved at the fourth attack of HZ. Multinucleated giant cells were determined on the Tzanck smear. VZV DNA was detected by polymerase chain reaction (PCR). Treatment with valacyclovir 1 g three times daily for 14 days was prescribed and then, prophylactic treatment with valacyclovir 500 mg two times a day was administered. Although immunosuppressive treatment was continued, no new attacks of herpes zoster occurred. We think that prophylactic antiviral therapy should be initiated in immunosuppressive individuals who have recurrent herpes zoster attacks.
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    Rekürren herpes zoster olgusunda profilaktik antiviral tedavi
    (Türkderm-Deri Hastalıkları ve Frengi Arşivi, 2011) Bayram, Hatice Gamze; Özcan, Hamdi; Bayındır, Yaşar
    Öz: Herpes zoster (HZ); sıklıkla yaşamın ilerleyen dönemlerinde, arka kök ganglionlarında eylemsiz halde bulunan varisella zoster virüsü (VZV)'nün aktifleşmesi ile ortaya çıkar. Bağışıklık sistemi baskılanmış hastalarda HZ insidansı 20-100 kat kadar artmıştır ve daha şiddetli seyretmektedir. Akut myelositik lösemi (AML) tip M3 tanısıyla immunsupresif tedavi uygulanan 48 yaşında erkek hasta polikliniğimize başvurdu. Hastaya klinik olarak HZ tanısı konuldu. Asiklovir 5x800 mg/gün ile HZ tedavisi yapıldı. Hasta ardışık olarak 3 ay üst üste benzer şikayetlerle tekrar başvurdu. Hastanın dördüncü herpes zoster atağında sol servikal (C)5, C6 dermatom alanları tutulmuştu. Tzanck yaymada multinükleer dev hücreler izlendi. Vezikülden alınan örnekte VZV DNA'sı polimeraz zincir reaksiyonu (PCR) pozitif bulundu. Valasiklovir 3x1 gr 14 gün süreyle uygulandı ve ardından valasiklovir 2x500 mg ile profilaktik tedaviye başlandı. Daha sonra immunsüpresif tedaviye davam edilmesine rağmen HZ atakları gözlenmedi. Bağışıklık sistemi baskılanmış bireylerde tekrarlayan HZ atağı gelişmesi durumunda profilaktik antiviral tedavi uygulaması gerektiğini düşünmekteyiz. (Türkderm 2011;45: 111-4) Başlık (İngilizce): Prophylactic antiviral treatment in recurrent herpes zoster: a case report Öz (İngilizce): Herpes zoster (HZ) occurs in older ages with activation of varicella-zoster virus (VZV) which persists in a dormant phase within the dorsal root ganglia. The incidence of HZ in immunosuppressed patients is 20-100 times higher and the clinical progress is more severe than in immunocompetent individuals. A 48-year-old man who had been diagnosed with acute myelocytic leukemia type M3 and had been treated with immunosuppressive agents was admitted to our clinic. The patient was clinically diagnosed as having HZ. He was treated with acyclovir 800 mg five times daily for 7 days. In the consecutive three months, he attended our clinic again with similar complaints. The left cervical (C)5, C6 dermatomes were involved at the fourth attack of HZ. Multinucleated giant cells were determined on the Tzanck smear. VZV DNA was detected by polymerase chain reaction (PCR). Treatment with valacyclovir 1 g three times daily for 14 days was prescribed and then, prophylactic treatment with valacyclovir 500 mg two times a day was administered. Although immunosuppressive treatment was continued, no new attacks of herpes zoster occurred. We think that prophylactic antiviral therapy should be initiated in immunosuppressive individuals who have recurrent herpes zoster attacks. 2 ATIF 11 Atıf Sayısı ui-button