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Öğe Bilateral episodic paroxysmal hemicrania: a case presentation(Springer Heidelberg, 2013) Kaplan, Y.; Kamisli, O.; Kamisli, S.; Altinayar, S.; Ozcan, C.[Abstract Not Available]Öğe The effect of oxcarbazepine and carbamazepine treatments on serum lipid levels(Dr Dietrich Steinkopff Verlag, 2008) Kurt, S.; Karaer, H.; Kaplan, Y.[Abstract Not Available]Öğe Evaluation of polymorphisms of the angiotensinogen M235T and angiotensin-converting enzyme gene insertion/deletion in patients craniocervical arterial dissection(Wiley-Blackwell, 2014) Celik, H.; Kaplan, Y.; Yesilada, E.; Gulbay, G.; Kamisli, O.; Ozcan, C.; Altinayar, S.[Abstract Not Available]Öğe Femoral nerve involvement in diabetics(Wiley, 2009) Kurt, S.; Kaplan, Y.; Karaer, H.; Erkorkmaz, U.In this study, the conduction of the femoral nerve has been evaluated in diabetic patients without clinical signs of femoral nerve involvement and in a group of healthy subjects. Forty-eight patients have been included in the study. Patients have been examined in terms of neuropathy and their neuropathy scores have been calculated. In addition to the nerve conduction studies have been performed. The findings of the diabetic patients have been compared with those of the 26 healthy volunteers. There has been a statistically significant difference between diabetics and the healthy volunteers in the control group in terms of both femoral nerve motor latency and amplitude. The femoral latencies of patients have significantly been related to the total neuropathy score. A significant difference between diabetic patients without polyneuropathy and the controls was observed with respect to their femoral latencies. In our study, femoral nerve conduction abnormalities have been determined in diabetics who clinically did not have femoral nerve involvement. It has been observed that these abnormalities become more evident as the polyneuropathy of the patients becomes more serious. Our study has shown that femoral nerve conductions may increase the sensitivity of the diagnosis of polyneuropathy.Öğe Gender differences in patients with total anterior circulation infarct(Wiley-Blackwell, 2012) Inanc, Y.; Kaplan, Y.; Kamisli, O.; Kamisli, S.; Ozcan, C.[Abstract Not Available]Öğe Increased mean platelet volume in cervicocephalic artery dissections(Springer London Ltd, 2012) Kamisli, O.; Kamisli, S.; Gonullu, S.; Kaplan, Y.; Ozcan, C.We aimed to investigate the role of platelets in the pathogenesis of cervicocephalic artery dissections by measuring the mean platelet volume (MPV) and platelet count. Thirty-four patients with evidence of cervicocephalic artery dissection were included in the study. Cervicocephalic artery dissection patients were divided into two subgroups as vertebral artery dissections and carotid artery dissections. There were 30 control subjects with similar baseline characteristics. The platelet count and MPV were compared between groups. The analysis showed a significant difference between the MPV values of the cervicocephalic artery dissection group and the control group (p < 0.05). However, when we compared subgroups with controls, we found a significantly higher MPV values only in the vertebral artery dissection group. We did not find any significant difference for total platelet count between the cervicocephalic artery dissection groups and controls. MPV is a marker of platelet function and activation. Increased MPV may be a result of dissection. In addition, it may be a risk factor for dissection-related stroke. Our study supports the notion that platelet reactivity is important in the pathogenesis of the cervicocephalic artery dissections, especially in vertebral artery dissections.Öğe Peduncular hallucinosis related with multiple sclerosis: a case report(Springer Heidelberg, 2012) Kamisli, O.; Kaplan, Y.; Kamisli, S.; Ozcan, C.[Abstract Not Available]Öğe Serum Gamma-glutamyltransferase Levels in Ischemic Stroke Subtypes(Karger, 2013) Kamisli, O.; Gonullu, S.; Kaplan, Y.; Kamisli, S.; Altinayar, S.; Ozcan, C.[Abstract Not Available]Öğe Transverse Sinus Thrombosis in a Living Donor: A Case Report(Elsevier Science Inc, 2013) Otan, E.; Aydin, C.; Karakas, S.; Yonder, H.; Kaplan, Y.; Yilmaz, S.Background. Liver transplantation is among the treatment options for end-stage liver disease. Limited organ donation in our country has resulted in an increased performance of living donor liver transplantations. This case report describes a left transverse cerebral venous sinus thrombosis diagnosed in a living donor hepatectomy patient. Patient. A 45-year-old man underwent right lobe hepatectomy of a 330-g graft for living donor liver transplantation to his 55-year-old hepatitis B virus positive brother. On the first postoperative day, without any surgical problems he presented with loss of consciousness. Previous medical history was unremarkable. Neurology consultation revealed lethargy and an acute confusional state. Cerebral magnetic resonance imaging venography showed signal alterations in the left transverse sinus wherein thrombosis was diagnosed. Heparin infusion initiated for antithrombotic treatment was adjusted to provide a 1.5 to 2-fold increased baseline activated partial thromboplastin time. On the second day of treatment has clinical status improved and he was discharged on the, fifth day on oral anticoagulant (warfarin) therapy for outpatient follow-up. Results. Cerebral venous sinus thrombosis is a rare disease with variable clinical onsets. Surgical procedures are among risk factors. An early diagnosis is essential to achieve low mortality and morbidity rates.Öğe Yawning headache associated with morbid obesity: presentation of two cases(Springer Heidelberg, 2012) Kaplan, Y.; Karaer, H.; Kurt, S.; Kamisli, O.[Abstract Not Available]
