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    Kafa travmaları I epidemiyoloji etiyoloji ve nörolojik değerlendirme
    (Sendrom, 2000) Serarslan, Yurdal; Çokluk, Cengiz; İyigün, Ömer Lütfi; Önder, Arif; Rakunt, Cemil; Çelik, Fahrettin
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    Kafa travmaları II sınıflama mekanizmalar ve fizyopatoloji
    (Sendrom, 2000) Serarslan, Yurdal; Çokluk, Cengiz; İyigün, Ömer Lütfi; Önder, Arif; Rakunt, Cemil; Çelik, Fahrettin
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    Kafa travmaları III radyolojik değerlendirme ve tedavi
    (Sendrom, 2000) Serarslan, Yurdal; Çokluk, Cengiz; İyigün, Ömer Lütfi; Önder, Arif; Rakunt, Cemil; Çelik, Fahrettin
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    The relationship between the neuron density of the trigeminal ganglion and the posterior communicating artery vasospasm in subarachnoid hemorrhage an experimental study
    (Neurosurgery Quarterly, 2012) Aydın, Mehmet Dumlu; Serarslan, Yurdal; Gündoğdu, Cemal; Aydın, Nazan; Aygül, Recep; Dündar, Dilcan Kotan; Hızır, Ulvi; Önder, Arif; Kanat, Ayhan
    Objective: Cerebral arteries innervated by several systems contribute to the control of cerebral blood flow. Sensory fibers of the trigeminal nerve have a vasodilatory effect on the basilar artery. Subarachnoid hemorrhage (SAH) causes severe cerebral vasospasm by various neurochemical mechanisms. We examined possible relationships between the neuron density of the trigeminal ganglion and the severity of basilar artery vasospasm in SAH. Methods: In this study, 28 rabbits were used. The animals were randomly divided into 3 groups: SAH (n=18), serum physiologic (n=5) and control (n=5) groups. Experimental SAH was induced by injecting homologous blood into the cisterna magna. After 20 days, the basilar arteries and trigeminal ganglions were examined histopathologically. Basilar artery volumes and the neuron density of the ophthalmic divisions of the trigeminal ganglions were estimated stereologically, and the results were statistically analyzed. Results: The mean basilar artery volume was 4.15±0.19 mm3 and the mean neuronal density of the trigeminal ganglion was 6500±750/mm3 for the control group. These values were 4.05±0.29 mm3 and 6400±584/mm3 for the serum physiologic group, 3.80±0.35 mm3 and 4600±300/mm3 for living animals in the SAH group, and 2.26±0.29 mm3 and 2950±618/mm3 for dead animals in SAH group, respectively. A linear relationship was found between the neuronal density of the trigeminal ganglion and basilar artery volumes. Conclusions: The neuron density of the trigeminal ganglion may be an important factor in the regulation of basilar artery volume and for the continuation of cerebral blood flow. The low neuron density of the trigeminal ganglion may be involved in the pathogenesis of severe basilar artery vasospasm induced by SAH. The Role of Trigeminal Ganglion Neuron Density in the Prevention of Subarachnoid Hemorrhage-induced Basilar Artery Vasospasm: An Experimental Study. Available from: https://www.researchgate.net/publication/232176472_The_Role_of_Trigeminal_Ganglion_Neuron_Density_in_the_Prevention_of_Subarachnoid_Hemorrhage-induced_Basilar_Artery_Vasospasm_An_Experimental_Study [accessed Jul 7, 2017].
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    Spinal arachnoiditis and axonal degeneration of the spinal nevre roots due to Ischemia induced by vasospasm an experimental study
    (Journal of Neurosurgical Sciences-Turkish, 2009) Önder, Arif; Serarslan, Yurdal; Aydın, Mehmet Dumlu; Kanat, Ayhan; Gürsan, Nesrin; Yolaş, Coşkun
    Various reasons have been ascribed to arachnoiditis development, among which subarachnoid hemorrhage (SAH) may be an important factor in this process. In this experimental study, we investigated the effect of SAH on histopathological findings. The volumetric changes of the radicular arteries and the density of spinal nerve root axons of a SAH model on C4 roots in rabbits were measured. Materials and Methods: In this study, 18 rabbits were used. The animals were randomly divided into three groups: subarachnoid hemorrhage (SAH; n = 10), physiologic serum (SF; n = 4) and control (n = 4) groups. SAH was performed by injecting 0.5 cc of blood into cisterna magna taken from their auricular veins. Cervical spinal nerve roots at the C4 level were examined histopathologically. Results: Meningeal thickening and adhesions, reddish spinal cord and radices were detected macroscopically. Histopathologically, leptomeningeal adhesions, intimal lesions of radicular arteries and axonal injury were detected at the nerve radices. The mean radicular artery volume was found to be low on the arachnoiditis developing animals. The mean alive axon density of the C4 nerve root decreased and axonal degeneration was observed in the SAH group. Conclusion: Our study suggests that SAH may be an important etiologic factor in spinal arachnoiditis.
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    Surgical management of the fourth ventricle arachnoid cysts
    (2019) Oral, Sukru; Bayarogullari, Hanifi; Yilmaz, Atilla; Urfali, Boran; Aras, Mustafa; Serarslan, Yurdal
    Aim: The arachnoid cysts are developmental masses that occur from the splitting or duplication of the arachnoid membrane. They may occur in different locations such as middle cranial fossa, retrocerebellar and convexity region, and they are usually asymptomatic. Arachnoid cysts are uncommon in association with intraventricular location especially fourth ventricle. In this study, we present six consecutive cases with arachnoid cyst in the fourth ventricle.Material and Methods: There were four female and two male patients. The average age of patients was 37 years ranging from2 to 65 years. These patients were periodically followed-up. The follow-up period ranged from minimum 2 to 6 years. Three patients were operated by a ventriculoperitoneal shunt and surgical excision was performed in two patients. Remaining two patients were followed up because they were asymptomatic.Results: Revision surgery was made due to shunt malfunction in a patient. The symptoms had been regressed in all patients. The arachnoid cysts were completely regressed after the operations in two patients.Conclusion: Most of arachnoid cysts which were small and asymptomatic did not require treatment. However, the size of an arachnoid cyst typically remained stable or increased over the time.An asymptomatic cyst may become symptomatic after minor head trauma. So, the asymptomatic patients with fourth ventricle arachnoid cyst should be under periodic follow up for obstructive hydrocephalus. Keywords: Arachnoid Cyst; Fourth Ventricle; Ventriculoperitoneal Shunt; Surgical Excision.