Iron Deficiency may be a Risk Factor for Inguinal Hernia Development in Children

Basit Öğe Kaydı
dc.authoridİnceoğlu, Feyza/0000-0003-1453-0937
dc.authorwosidİnceoğlu, Feyza/GVK-2847-2022
dc.contributor.authorHarma, Birsen
dc.contributor.authorKiran, Tugba Raika
dc.contributor.authorInceoglu, Feyza
dc.date.accessioned2024-08-04T20:59:46Z
dc.date.available2024-08-04T20:59:46Z
dc.date.issued2023
dc.departmentİnönü Üniversitesien_US
dc.description.abstractObjectives: This case control study aimed to investigate whether the routine hemogram and biochemical parameters of pediatric patients who have undergone surgery for inguinal hernia are associated with the etiopathogenesis of the disease. Methods: Eighty cases of inguinal hernia surgery performed between January 2019 and November 2022 were included in the study. A control group was also established using hospital records, consisting of eighty pediatric patients without any known his-tory of hematological or metabolic disease or use of regular medication. Statistical analysis was conducted to compare the total hemoglobin (Hgb), hematocrit (Htc), mean corpuscular volume (MCV), mean corpuscular hemoglobin (MCH), mean corpuscular hemoglobin concentration (MCHC), erythrocyte distribution width (RDW) and thrombocyte (PLT) values in both groups. Results: The age range of the pediatric patients was 1-14 years. Of the eighty children, 47 (58.8%) were male and 33 (41.3%) were female, with a mean age of 5.79 +/- 3.26. The values of Hgb, Htc, MCH, MCHC, and MCV in the inguinal hernia patients were found to be statistically significantly lower than those in the control group (p<0.05). Additionally, the patient RDW values were found to be statistically significantly higher than those in the control group (p<0.05). Conclusion: Compared to the control group, the observed decrease in MCH, MCHC, MCV, Hgb, HTC values, as well as the increase in RDW in patient group, suggests a predisposing effect of iron deficiency. These specific changes suggested that iron deficiency may lead structural changes in the collagen construction and may contribute the etiopathogenesis of childhood inguinal hernia.en_US
dc.identifier.doi10.14744/SEMB.2023.60343
dc.identifier.endpage110en_US
dc.identifier.issn1302-7123
dc.identifier.issn1308-5123
dc.identifier.issue1en_US
dc.identifier.pmid37064850en_US
dc.identifier.startpage105en_US
dc.identifier.urihttps://doi.org/10.14744/SEMB.2023.60343
dc.identifier.urihttps://hdl.handle.net/11616/103504
dc.identifier.volume57en_US
dc.identifier.wosWOS:000980488900013en_US
dc.identifier.wosqualityQ3en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherKare Publen_US
dc.relation.ispartofMedical Bulletin of Sisli Etfal Hospitalen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectChilden_US
dc.subjectErythrocyte Distribution Width (RDW)en_US
dc.subjectinguinal herniaen_US
dc.subjectiron deficiencyen_US
dc.titleIron Deficiency may be a Risk Factor for Inguinal Hernia Development in Childrenen_US
dc.typeArticleen_US

Dosyalar

Koleksiyon

WoS İndeksli Yayınlar Koleksiyonu
PubMed İndeksli Yayın Koleksiyonu