Hepatit A Enfeksiyonu Sonrası Gelişen Bir Guillian-Barre Sendromu Olgusu+
| dc.contributor.author | Özışık, Handan Işın | |
| dc.contributor.author | Kızkın, Sibel | |
| dc.contributor.author | Çalışkan, Özden | |
| dc.contributor.author | Özcan, Cemal | |
| dc.date.accessioned | 2015-04-16T13:09:21Z | |
| dc.date.available | 2015-04-16T13:09:21Z | |
| dc.date.issued | 2002 | |
| dc.description | İnönü Üniversitesi Tıp Fakültesi Dergisi 9(2) 131-133 (2002) | en_US |
| dc.description.abstract | Guillian-Barre Syndrome (GBS) is often preceded by an infectious disease. A case of GBS after hepatitis A (HA) is extremely rare, even though hepatitis A is common. In a review of case reports the clinical features of GBS following HA could be sumarized as follows: 1-Most of the patients are men 2- GBS develops within 14 days after onset of HA 3- Facial nerve and bulbus are effected frequently 4- Proprioception is likely impaired in addition to superficial sensation 5- Mechanic ventilation is often requirement 6- The protein content of the cerebrospinal fluid is usually high 7- The outcome of neuropathic symptoms is uniformly good, regardless of the degree of liver dysfunction as evaluated on the basis of alanine aminotransferase levels 8- Patient is frequently recover without sequelaw 9- ENMG demonstrate demyelinations. These findings indicate that GBS following HA essentially does not differ from typical GBS. | en_US |
| dc.description.abstract | Guillian-Barre Syndrome (GBS) is often preceded by an infectious disease. A case of GBS after hepatitis A (HA) is extremely rare, even though hepatitis A is common. In a review of case reports the clinical features of GBS following HA could be sumarized as follows: 1-Most of the patients are men 2- GBS develops within 14 days after onset of HA 3- Facial nerve and bulbus are effected frequently 4- Proprioception is likely impaired in addition to superficial sensation 5- Mechanic ventilation is often requirement 6- The protein content of the cerebrospinal fluid is usually high 7- The outcome of neuropathic symptoms is uniformly good, regardless of the degree of liver dysfunction as evaluated on the basis of alanine aminotransferase levels 8- Patient is frequently recover without sequelaw 9- ENMG demonstrate demyelinations. These findings indicate that GBS following HA essentially does not differ from typical GBS. | en_US |
| dc.identifier.citation | Özışık, Handan Işın ;Kızkın, Sibel ; Çalışkan, Özden ; Özcan, Cemal ;İnönü Üniversitesi Tıp Fakültesi Dergisi 9(2) 131-133 (2002) | en_US |
| dc.identifier.uri | http://www.totmdergisi.org/articles/2002/volume9/issue2/2002_9_2_12.pdf | |
| dc.identifier.uri | https://hdl.handle.net/11616/1875 | |
| dc.language.iso | tr | en_US |
| dc.publisher | İnönü Üniversitesi Tıp Fakültesi Dergisi | en_US |
| dc.rights | Attribution 3.0 United States | * |
| dc.rights.uri | http://creativecommons.org/licenses/by/3.0/us/ | * |
| dc.subject | Hepatit A | en_US |
| dc.subject | Guillian-Barre Sendromu | en_US |
| dc.subject | GBS Klinik Bulguları | en_US |
| dc.title | Hepatit A Enfeksiyonu Sonrası Gelişen Bir Guillian-Barre Sendromu Olgusu+ | en_US |
| dc.title.alternative | A Case Of Guillian-Barre Syndrome After Acute Viral Hepatitis A | en_US |
| dc.type | Article | en_US |
